Biography
Joon-Young Park is an associate professor of kinesiology in the College of Public Health at Temple University. He is a professional member of American Physiological Society, American Heart Association and American College of Sports Medicine. He previously has served or currently serves as an editorial board member for several scientific journals including Exercise and Sport Science Reviews, Clinical Science, Integrative Medicine Research, and Journal of Exercise Rehabilitation. He is a recipient of the Scientist Development Grant Award and Certificate of Research Excellence Award from the American Heart Association. He is the author or co-author of more than 30 peer-reviewed articles, over 60 refereed conference proceedings and four book chapters.
Dr. Park's current research focuses on determining the molecular basis of exercise effects on vascular disease and function. Exercise is the most potent nonpharmacological interventions for cardiovascular diseases. Understanding the fundamental molecular mechanisms of the exercise effects is crucial as it can provide insights into the pathological processes during disease development and may lead to the discovery of novel therapeutic targets. Current research projects focus on answering such questions as: How can the physiological changes during exercise be defined at the cellular level? How do endothelial cells respond to those associated stimuli? What mediates cellular responses to the stimulus? And, how do cells adapt to the stimulus when it persists repeatedly and chronically? His teaching includes courses in the context of integrative exercise physiology in cardiovascular, neuromuscular and metabolic systems.
Courses Taught
Number | Name | Level |
|---|---|---|
HRPR 5999 | Research Experience in Health Professions | Graduate |
KINS 9201 | Cardiovascular Exercise Physiology | Graduate |
KINS 9203 | Appl Ex Physio-Neuromusc | Graduate |
KINS 9204 | Cellular Adapt to Exer | Graduate |
Selected Publications
Recent
Kim, J., Sayoc, J., Baek, K., & Park, J. (2021). Laminar shear stress protects against premature endothelial senescence by sirt1-dependent mechanisms. Exercise Science, 30(2), 213-220. doi: 10.15857/KSEP.2021.30.2.213.
Hong, J., Hong, S., Lee, J., Park, J., Eriksen, J., Rooney, B., & Park, Y. (2020). Exercise training ameliorates cerebrovascular dysfunction in a murine model of Alzheimer's disease: Role of the P2Y2 receptor and endoplasmic reticulum stress. American Journal of Physiology - Heart and Circulatory Physiology, 318(6), H1559-H1569. doi: 10.1152/AJPHEART.00129.2020.
Kim, T., Park, S., Park, J., & Park, H. (2020). Infusion of plasma from exercised mice ameliorates cognitive dysfunction by increasing hippocampal neuroplasticity and mitochondrial functions in 3xtg-ad mice. International Journal of Molecular Sciences, 21(9). doi: 10.3390/ijms21093291.
Tian, R., Colucci, W., Arany, Z., Bachschmid, M., Ballinger, S., Boudina, S., Bruce, J., Busija, D., Dikalov, S., Dorn, G., Galis, Z., Gottlieb, R., Kelly, D., Kitsis, R., Kohr, M., Levy, D., Lewandowski, E., McClung, J., Mochly-Rosen, D., O'Brien, K., O'Rourke, B., Park, J., Ping, P., Sack, M., Sheu, S., Shi, Y., Shiva, S., Wallace, D., Weiss, R., Vernon, H., Wong, R., & Longacre, L.S. (2019). Unlocking the Secrets of Mitochondria in the Cardiovascular System: Path to a Cure in Heart Failure—A Report from the 2018 National Heart, Lung, and Blood Institute Workshop. Circulation, 140(14), 1205-1216. doi: 10.1161/circulationaha.119.040551.
Joo, H., Park, J., & Rhyu, H. (2019). Effects of the senior welfare center exercise program on body shape, physical fitness level, and cardiovascular health-related factors in old man from Korean rural areas. Journal of Exercise Rehabilitation, 15(2), 282-286. doi: 10.12965/jer.1938072.036.
Kim, J. & Park, J. (2019). Effects of resveratrol on laminar shear stress-induced mitochondrial biogenesis in human vascular endothelial cells. Physical Activity and Nutrition, 23(1), 7-12. doi: 10.20463/jenb.2019.0002.
Kokkinaki, D., Hoffman, M., Kalliora, C., Kyriazis, I., Maning, J., Lucchese, A., Shanmughapriya, S., Tomar, D., Park, J., Wang, H., Yang, X., Madesh, M., Lymperopoulos, A., Koch, W., Christofidou-Solomidou, M., & Drosatos, K. (2019). Chemically synthesized Secoisolariciresinol diglucoside (LGM2605) improves mitochondrial function in cardiac myocytes and alleviates septic cardiomyopathy. Journal of Molecular and Cellular Cardiology, 127, 232-245. doi: 10.1016/j.yjmcc.2018.12.016.
Salamah, A., Shin, J., & Park, J. (2019). Effect of NSAIDs on Muscle Adaptations to Resistance Training: A Brief Review. Journal of Public Health Issues and Practices, 3(1). doi: 10.33790/jphip1100132.
Lee, H., Kim, K., Kim, B., Shin, J., Rajan, S., Wu, J., Chen, X., Brown, M., Lee, S., & Park, J. (2018). A cellular mechanism of muscle memory facilitates mitochondrial remodelling following resistance training. Journal of Physiology, 596(18), 4413-4426. doi: 10.1113/JP275308.
Cheng, Z., Shen, X., Jiang, X., Shan, H., Cimini, M., Fang, P., Ji, Y., Park, J., Drosatos, K., Yang, X., Kevil, C., Kishore, R., & Wang, H. (2018). Hyperhomocysteinemia potentiates diabetes-impaired EDHF-induced vascular relaxation: Role of insufficient hydrogen sulfide. Redox Biology, 16, 215-225. doi: 10.1016/j.redox.2018.02.006.
Zeng, H., Nanayakkara, G., Shao, Y., Fu, H., Sun, Y., Cueto, R., Yang, W., Yang, Q., Sheng, H., Wu, N., Wang, L., Yang, W., Chen, H., Shao, L., Sun, J., Qin, X., Park, J., Drosatos, K., Choi, E., Zhu, Q., Wang, H., & Yang, X. (2018). DNA checkpoint and repair factors are nuclear sensors for intracellular organelle stresses-inflammations and cancers can have high genomic risks. Frontiers in Physiology, 9(MAY). doi: 10.3389/fphys.2018.00516.
Shao, Y., Nanayakkara, G., Cheng, J., Cueto, R., Yang, W., Park, J., Wang, H., & Yang, X. (2018). Lysophospholipids and their receptors serve as conditional DAMPs and DAMP receptors in tissue oxidative and inflammatory injury. Antioxidants and Redox Signaling, 28(10), 973-986. doi: 10.1089/ars.2017.7069.
Seki, M., Powers, J., Maruyama, S., Zuriaga, M., Wu, C., Kurishima, C., Kim, L., Johnson, J., Poidomani, A., Wang, T., Muñoz, E., Rajan, S., Park, J., Walsh, K., & Recchia, F. (2018). Acute and chronic increases of circulating FSTL1 normalize energy substrate metabolism in pacing-induced heart failure. Circulation: Heart Failure, 11(1). doi: 10.1161/CIRCHEARTFAILURE.117.004486.